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REFERENSER 1. Wolever TMS, Bjrck I, Granfeldt Y, Xiaomei W, Brand-Miller J, Brighenti F, Holt S, Mann J, Perry TL, Venter C, Vorster H: Determination of the glycemic index of foods: Interlaboratory study. Manuscript ; 2. FAO WHO: Carbohydrates in human nutrition: report of a joint FAO WHO expert consultation. FAO Food and Nutrition Paper 66 1998: 1-140. Slabber M, Barnard HC, Kuyl J.M, Dannhauser A, Schall R: Effects of a low-insulin-response, energy-restricted diet on weight loss and plasma insulin concentrations in hyperinsulinemic obese females. J Clin Nutr 1994; 60: 48-53. Foster-Powell K, Brand Miller J: International tables of glycemic index. J Clin Nutr 1995; 62: 871S-893S. Liljeberg H, Bjrck I: Delayed gastric emptying rate as a potential mechanism for lowered glycemia after eating sourdough bread: studies in humans and rats using test products with added organic acids or an organic salt. J Clin Nutr 1996; 64: 886-93. de Boer S, Masclee A, Lam W et al: Hyperglycaemia modulates gallbladder motility and small intestinal transit time in man. Dig Dis Sci 1993; 38: 2228-35. Jones K, Doran S, Hveem K, Bartholomeusz F, Morley J, Sun W-M: Relationship between satiation and antral area in normal subjects. J Clin Nutr 1997; 66: 127-32. Lavin J, Wittert G, Sun W-M, Horowitz M, Morley J, Read N: Appetite regulation by carbohydrates: role of blood glucose and gastrointestinal hormones. J Physiol 1996; 271: E209-14. 9. Lavin J, Wittert G, Andrews J et al: Interaction of insulin, glucagonlike peptide-1, gastric inhibitory peptide and appetite in response to intraduodenal carbohydrate. J Clin Nutr 1998; 68: 591-8. Bjrck I, Liljeberg H, stman E: Low glyca. Administration of anti-RhD by the intramuscular route, either MonoRho or Rhophylac resulted in a slower saturation of binding sites reaching levels of 20-67% after 48 hours Figure 4 ; . However, this did not jeopardize removal of RhD positive RBC as clearance began when approx. 20% of the binding sites displayed bound anti-RhD, data not shown. Fc receptor analysis The majority of subjects were typed for the Fc RIIA-131H R, Fc IIIA-158V F, and Fc RIIIB NA1 NA2 polymorphisms as follows; 23 subjects receiving MonoRho intravenous; 6 receiving MonoRho intramuscular; 7 out of 9 receiving Rhophylac intravenous and 6 receiving Rhophylac intramuscular Table 1 ; . The MonoRho intravenous data revealed an association of Fc RIIA p 0.05 ; and IIIA p 0.05 ; allotypes on the RBC clearance rate while no dependence on Fc RIIIB polymorphisms was seen p 0.87 ; Figure 5a, b, c ; . The borderline degree of significance obtained for Fc RIIA and Fc RIIIA using this rigorous analysis is related to the small sample size but a clear trend is shown Figure 5 ; . Subjects homozygous for Fc RIIA-131H or Fc RIIIA-158V allotypes showed a faster clearance rate compared to both the heterozygote Fc RIIA-H R, Fc RIIIA-V F and the alternative homozygote Fc RIIA-RR and Fc RIIIA-FF. The RBC clearance rates for subjects receiving Rhophylac intravenous were all fast and no statistical analysis could be performed Table 1 ; . Administration of anti-RhD by the intramuscular route resulted in slower RBC clearance rates compared to the intravenous route. Despite the smaller number of subjects in the intramuscular groups , a similar but not so marked trend of correlation with Fc R polymorphisms was seen Table 1 ; . In both the Rhophylac and MonoRho intramuscular treatment groups the presence of Fc RIIA-131R and Fc RIIIA-158F was correlated with a slower removal of antibody coated cells; for example, the 2 subjects no's. 1005 and 1006 ; homozygous for both allotypes IIA-RR and IIIA-FF had the longest RhD positive RBC elimination half-lives 50.3 and 131.4 h ; Table 1 ; . In summary, these results showed that for both polyclonal and recombinant antibodies there were faster RBC clearance rates after intravenous administration than intramuscular. In particular, for MonoRho administered intravenous the RBC clearance rate was not dependent on the dose of anti-RhD used but instead showed an interesting association with the Fc RIIA and IIIA polymorphisms.

From the Division of Hematology and Internal Medicine A.D., R.A.K., M.Q.L., S.V.R., R.F., P.R.G., T.E.W., J.A.L., S.R.Z, D.S.S., S.R.H., R.S.A., M.R.L., D.A.G., A.T., D.J.I., I.M.M., S.M.A., L.F.P., M.A.E., and M.A.G. ; , the Division of Biostatistics T.M.T., D.R.L., and M.F.P. ; . Mayo Clinic, Rochester, MN. Corresponding author and reprint requests: Angela Dispenzieri, M.D. Mayo Clinic, 200 First Street S.W., Rochester, MN 55905, USA; Telephone 507 284-2479; Fax: 507 266-4972; e-mail: dispenzieri.angela mayo. Ted form the original scaling is one of the reasons for the increased distributed power. An increase of the number of transistors in a chip by more than factor of K 2 another reason for the power increase. In fact, the transistor size decreased by a factor of 0.7 while the transistor area decreased by factor of 0.5 for every generation. Present complex digital designs cannot wait for the downscaling and thus the actual chip size increased by a factor of four, more than predicted by standard scaling rules. Introducing new technologies such as multilayer interconnections, double polysilicon and further complicated cell structures for the memories partially solves the problem of insufficient IC area. Recent progress in the CMOS scaling has been achieved using improved DUV lithography tools. Originally, targeted at the 0.35 m devices, these tools were successfully introduced at 0.25 m level and are being used in the current 0.18 m generation. The use of these tools is projected at least for 0.15 m devices. Further progress is required to adapt popular reticle enhancement technique RETs ; , proximity correction and phase shift mask OPC PSM ; to obtain improved packaging densities. From the extrapolation of traditional scaling, UDSM devices are expected to have excellent drive current and the projected performance suggests circuits operating at frequencies up to 10 GHz. On the other hand, one should remember that there are serious technological and economic limitations of further, accelerated improvements of the standard CMOS technologies and the transistor performance could be, to some extent, compromised. In aggressively sized technologies, oxide scaling leads to rapidly increased gate currents, regardless of the oxide quality. Further improvements in the reduction of the gate tunneling currents require the use of alternative gate dielectric materials. High-k materials are good candi32 and benzphetamine.

Table 1. Effects of various concentrations of ethanol in vitro on purified glucose 6-phosphate dehydrogenase G6PD ; from human erythrocyte Ethanol concentration mM ; 0 drugless ; a 0.242 0.484 0.968 G6PD activity 100 0 90 0 and benztropine.

We have shown for the first time that both isoforms of human DNA topo II are targeted by etoposide in vivo. This report describes a novel "TARDIS" method with which etoposide-induced topo II - and -cleavable complex formation in individual cells can be visualized and quantified. Using this method, we have shown that both isoforms form cleavable complexes in vivo after etoposide treatment in a dose dependent manner. This confirms the previously obtained in vitro data Cornarotti et al., 1996; Marsh et al., 1996 ; and data from a yeast model system Meczes et al., 1997 ; , which indicated that both isoforms were potential targets for etoposide. The results showed that the increase in immunofluorescence intensity was dependent on etoposide concentration. If there is one sure way of extending the life of machinery, gearboxes and hydraulic systems, it is by using clean, uncontaminated oil. CC Jensen will be showing a range of filters, off-line fine and bepridil.
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